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Hibiscus Oil -(Hibiscus Sabdariffa L)- Essential Oil 100% Pure Natural Undiluted Uncut Therapeutic Grade Oil 1.01 Fl.OZ

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Hibiscus seed has a golden yellow color, and as with most oils I’ve worked with, the color varies slightly depending on the source and level of refinement. Hibiscus oil has a balanced fatty acid profile that leans towards being more moisturizing and better for drier skin types due to higher levels of oleic acid. The Oleic acid content usually ranges 35-40% and is well-balanced by 25-35% linoleic acid. Having these two critical fatty acids balanced in that proportion results in an oil that's anti-inflammatory as well as moisturizing while being very unlikely to clog pores. Will Hibiscus Oil Clog Pores When my skin is dry, it gets very itchy. Hibiscus oil prevents the itchiness and burning associated with winter dryness. Interestingly, studies have shown that hibiscus may promote liver health and help keep your liver working efficiently.

Panieri E, Santoro MM. ROS homeostasis and metabolism: a dangerous liason in cancer cells. Cell Death Dis. 2016;7(6):e2253. Published 2016 Jun 9. https://doi.org/10.1038/cddis.2016.105.Hibiscus is an ultra-hydrating and nourishing herb that is a rich source of protein, calcium, copper, iron, magnesium, manganese, potassium, zinc, vitamins-A, B6, C, E, and K. Besides, it also contains niacin, thiamin, riboflavin, antioxidants, alpha hydroxy acids, beta hydroxy acids, malic acid, and many other compounds that benefit your skin. [ 1]

Another test-tube study reported that hibiscus leaf extract prevented human prostate cancer cells from spreading ( 22).Besides antioxidant activity, the compounds in hibiscus may also provide benefits through other mechanisms that are not as well understood. Again, more research is needed to learn more. Summary We have investigated aqueous hibiscus flower extract anticancer efficacy, selectivity, and interactions with chemotherapeutics taxol, cisplatin, and tamoxifen in estrogen-receptor positive breast cancer cells, triple-negative human breast cancer cells, and normal non-cancerous cells. Apoptotic morphology and biochemical marker expression were assessed to determine the extent anticancer efficacy of hibiscus. Mitochondrial membrane potential reduction and reactive oxygen species generation were quantified using fluorogenic dyes to determine the mechanism of hibiscus extract action. Results All varieties of carrier oils have many overlapping benefits for the skin. Hibiscus oil skin benefits include: Hibiscus aids in healing wounds faster by increasing the production of fibronectin protein in your skin. It also boosts hydration and cell regeneration in your skin which play a significant role in the process of wound healing. Annexin V binding assay and propidium iodide staining were performed to respectively monitor early apoptosis and cell permeabilization, a marker of necrotic or late apoptotic cell death. Cells were treated with various concentrations of hibiscus flower extract similar to those published previously with aqueous extracts of dandelion root and white tea [ 18, 19]. Cells were then treated individually or in combination with chemotherapeutics taxol, cisplatin, and tamoxifen as indicated in the results section. This protocol is similar to that previously published [ 18, 19]. Cells were washed with phosphate-buffered saline (PBS) and suspended in Annexin V binding buffer (10 mM HEPES, 140 mM NaCl, 2.5 mM CaCl 2, pH 7.4) with green fluorescent Annexin V AlexaFluor-488 (1:20) (Life Technologies Inc., Burlington, ON, CA, Cat No. A13201) and 0.01 mg/mL of red fluorescent PI (Life Technologies Inc., Burlington, ON, CA, Cat No. P3566) for 15 min at 37 °C protected from light. Percentage of early (green), late apoptotic cells (green and red), and necrotic cells (red) were quantified with a Tali Image-Based Cytometer (Life Technologies Inc., Burlington, ON, CA, Cat No. T10796). Cells from at least 18 random fields were analyzed using both the green (ex. 458 nm; em. 525/20 nm) and red (ex. 530 nm; em. 585 nm) channels. Fluorescent micrographs were taken at 400x magnification using LAS AF6000 software with a Leica DMI6000 fluorescent microscope (Wetzlar, Germany). Cells monitored with microscopy were counterstained with Hoechst 33342 (Molecular Probes, Eugene, OR, USA) with a final concentration of 10 μM during the 15-min incubation. Reactive oxygen species (ROS) quantification

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